Author

K. F. Wilson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

R. A. Cerione· 2 shared A. August· 1 shared D. Argueta-Zamora· 1 shared I. Y. Hwang· 1 shared J. C. Sanchez· 1 shared K. S. Green· 1 shared K. S. Rojas· 1 shared M. R. Zanotelli· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCancer Biology

R. A. Cerione

Mutual co-authors in cluster

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2 papers

Recent·Most cited

PTEN loss in glioma cell lines leads to increased extracellular vesicle biogenesis and PD-L1 cargo in a PI3K-dependent manner

J. C. Sanchez, T. M. Pierpont, D. Argueta-Zamora, K. F. Wilson, A. August, R. A. Cerione

This study shows PTEN loss increases EV biogenesis and PD-L1 cargo in glioma cells via PI3K signaling, enhancing immunosuppression.

bioRxiv · Jul 26, 2023Cancer Biology

A multiprotein signaling complex sustains AKT and mTOR/S6K activity necessary for the survival of cancer cells undergoing stress

O. Y. Teran Pumar, M. R. Zanotelli, M.-c. J. Lin, R. R. Schmitt, K. S. Green, K. S. Rojas, I. Y. Hwang, R. A. Cerione, K. F. Wilson

This study identifies a multiprotein complex, DockTOR, that sustains AKT and mTOR/S6K activity in cancer cells under stress, enabling survival through a mechanism independent of growth factor signaling.

bioRxiv · Jan 3, 2023 · PreprintCancer Biology