Author

K. Pervolaraki

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Organism:mouse· 1

Frequent Co-Authors

D. Albrecht· 2 shared S. Boulant· 2 shared C. Bamford· 1 shared C. Odendall· 1 shared F. Bormann· 1 shared H. Ohno· 1 shared J. Kagan· 1 shared J. McLauchlan· 1 shared

Activity by Year

2018-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersImmunology

D. Albrecht

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2 papers

Recent·Most cited

Cellular polarity asymmetrically functionalizes pathogen recognition receptor-mediated intrinsic immune response in human intestinal epithelium cells

M. Stanifer, S. Muenchau, K. Pervolaraki, T. Kanaya, M. Mukenhirn, D. Albrecht, C. Odendall, J. Kagan, S. Bartfeld, H. Ohno, S. Boulant

The paper shows that AP-1B-mediated polarized sorting of TLR3 leads to an asymmetric immune response in human intestinal epithelial cells to basolateral vs. apical infections.

bioRxiv · Nov 7, 2019 · PreprintImmunology

Differential induction of interferon stimulated genes between type I and type III interferons is independent of interferon receptor abundance

K. Pervolaraki, S. Rastgou Talemi, D. Albrecht, F. Bormann, C. Bamford, J. Mendoza, K. C. Garcia, J. McLauchlan, T. Höfer, M. L. Stanifer, S. Boulant

This paper shows that type I and III interferons induce interferon stimulated genes with different kinetics, and these differences are intrinsic to the signaling pathways, not receptor abundance.

bioRxiv · Oct 19, 2018Immunology