Author

K. Verma

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Organism:E. coli· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. Chaphalkar· 2 shared K. Chakraborty· 2 shared A. Dowell· 1 shared A. Horsley· 1 shared A. Shukla· 1 shared B. Hallis· 1 shared B. Parker· 1 shared C. Sachidanandan· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersBiochemistry

A. Chaphalkar

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Live visualization of endoplasmic reticulum redox potential in zebrafish embryos reveals region-specific differences

M. Verma, N. R. Bhatt, A. Chaphalkar, K. Verma, S. Umale, S. Verma, C. Sachidanandan, K. Chakraborty

This study uses transgenic zebrafish to visualize ER redox potential in live embryos, revealing region-specific deviations from expected oxidizing states.

bioRxiv · Feb 12, 2022Biochemistry

Robust SARS-CoV-2-specific T-cell immunity is maintained at 6 months following primary infection

J. Zuo, A. Dowell, H. Pearce, K. Verma, H. Long, J. Begum, F. Aiano, Z. Amin-Chowdhury, B. Hallis, L. Stapley, R. Borrow, E. Linley, S. Ahmad, B. Parker, A. Horsley, , P. Moss

This study shows that SARS-CoV-2-specific T-cell immunity remains robust six months after infection, with response magnitude linked to initial infection severity.

bioRxiv · Nov 1, 2020Immunology

Distinct metabolic states of a cell guide alternate fates of mutational buffering through altered proteostasis

K. Verma, K. Saxena, R. Donaka, A. Chaphalkar, M. K. Rai, A. Shukla, Z. Zaidi, R. Dandage, D. Shanmugam, K. Chakraborty

The paper shows that metabolic states in E. coli influence proteostasis and mutational buffering through metabolite-dependent chaperone assistance, not increased chaperone levels.

bioRxiv · Mar 20, 2020Biochemistry