Author

Kaval Reddy Prasasvi

Indian Institute of Science Bangalore

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:deep learning· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Abhishek ChowdhuryiD· 3 shared Kumaravel SomasundaramiD· 3 shared Lakshay Garg· 3 shared Subhrodeep SahaiD· 2 shared Anand Kumar SrivastavaiD· 1 shared Arani Mukherjee· 1 shared Bidisha Prajapati· 1 shared Dhrubjyoti SharmaiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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3 shared papersCancer Biology

Abhishek ChowdhuryORCID

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3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJul 16, 2026

Seizure-related gene 6 (SEZ6) encodes a Cancer stem cell-specific proangiogenic molecule: A novel glioma therapeutic target

Arani Mukherjee, Shreoshi Sengupta, Abhishek Chowdhury…·Kumaravel Somasundaram

This study identifies SEZ6 as a glioma stem cell-specific proangiogenic molecule that promotes angiogenesis through TGFβ signaling, offering a novel therapeutic target for glioblastoma.

DOI 10.64898/2026.07.15.738615

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivPreprintNov 20, 2025

Calcitonin-Driven Reactivation of the Hippo Tumor-Suppressor Cascade Attenuates YAP/TAZ Oncogenic Signaling in Glioblastoma

Jayita Goswami, Prithviraj Uttarasili, Lakshay Garg…·Kumaravel Somasundaram

This study shows that calcitonin activates the Hippo pathway via CTR to inhibit YAP/TAZ in glioblastoma, with structural defects in CTR mutants impairing this effect.

DOI 10.1101/2025.11.20.689524

humanproteomics

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Cancer BiologybioRxivPreprintNov 20, 2024

BAZ1A, an Imitation Switch (ISWI) protein, interacts and facilitates the recruitment of E2F1 to activate the E2F transcription program

Bidisha Prajapati, Abhishek Chowdhury, Kaval Reddy Prasasvi…·Kumaravel Somasundaram

This study identifies BAZ1A as a key component of an ISWI complex that recruits E2F1 to activate the E2F transcription program, promoting G1-S progression in glioma cells.

DOI 10.1101/2024.11.20.624462

humanbulk RNA-seq

𝕏 PostBluesky