The pivotal role of a novel free fatty acid receptor GPR164 in the intestinal barrier function
Takako Ikeda, Yuki Masujima, Keita Watanabe, Akari Nishida, Mayu Yamano, Miki Igarashi, Nobuo Sasaki, Hironori Katoh, Ikuo Kimura
This study shows that GPR164 deletion in mice causes intestinal barrier dysfunction, Wnt/β-catenin signaling disruption, and IBD-like symptoms, suggesting GPR164 as a therapeutic target for IBD.
bioRxiv · Feb 20, 2025 · PreprintPhysiology