Author

Hironori Katoh

Osaka Metropolitan UniversityORCID 0000-0002-8191-8117Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

Akari NishidaiD· 1 shared Ikuo KimuraiD· 1 shared Keita Watanabe· 1 shared Mayu YamanoiD· 1 shared Miki IgarashiiD· 1 shared Nobuo SasakiiD· 1 shared Takako IkedaiD· 1 shared Yuki Masujima· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 28 mutual co-author links

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1 shared paperPhysiology

Akari NishidaORCID

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1 paper

Recent·Most cited

The pivotal role of a novel free fatty acid receptor GPR164 in the intestinal barrier function

Takako Ikeda, Yuki Masujima, Keita Watanabe, Akari Nishida, Mayu Yamano, Miki Igarashi, Nobuo Sasaki, Hironori Katoh, Ikuo Kimura

This study shows that GPR164 deletion in mice causes intestinal barrier dysfunction, Wnt/β-catenin signaling disruption, and IBD-like symptoms, suggesting GPR164 as a therapeutic target for IBD.

bioRxiv · Feb 20, 2025 · PreprintPhysiology