Author

Kyle Burrows

Primary ORCID profile (0000-0001-5371-3512 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Kyle BurrowsiD1 paper · Simon Fraser University

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

Bruce Vallance· 1 shared Colby Zaph· 1 shared Håvard T. LindholmiD· 1 shared John Arne DahliD· 1 shared Madeleine FosslieiD· 1 shared Mara Martín-AlonsoiD· 1 shared Menno J. OudhoffiD· 1 shared Naveen Parmar· 1 shared

Activity by Year

2020-2020 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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1 shared paperImmunology

Bruce Vallance

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1 paper

Recent·Most cited

Intestinal-epithelial LSD1 controls cytoskeletal-mediated cell identity including goblet cell effector responses required for gut inflammatory and infectious diseases

Naveen Parmar, Kyle Burrows, Håvard T. Lindholm, Rosalie T. Zwiggelaar, Mara Martín-Alonso, Madeleine Fosslie, Bruce Vallance, John Arne Dahl, Colby Zaph, Menno J. Oudhoff

This study shows that intestinal-epithelial LSD1 deletion leads to increased pathogenesis in inflammatory and infectious disease models by impairing cytoskeletal organization and goblet cell effector responses.

bioRxiv · Jul 9, 2020Immunology