Author

Madeleine Fosslie

Oslo University HospitalORCID 0000-0002-2902-4394Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Organism:mouse· 3 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

John Arne DahliD· 3 shared Håvard T. LindholmiD· 2 shared Mara Martín-AlonsoiD· 2 shared Menno J. OudhoffiD· 2 shared Naveen Parmar· 2 shared Rosalie T. ZwiggelaariD· 2 shared Adeel ManafiD· 1 shared Alberto Díez-SáncheziD· 1 shared

Activity by Year

2020-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersDevelopmental Biology

John Arne DahlORCID

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3 papers

Recent·Most cited

Major waves of H2A.Z incorporation during mouse oogenesis

Madeleine Fosslie, Erkut Ilaslan, Trine Skuland, Michel Choudalakis, M. Dige Soegaard, Jason Alexander Halliwell, Shaista Khan, Marie Indahl, María Vera-Rodríguez, Adeel Manaf, Annika V Geijer-Simpson, Rajikala Suganthan, Ingunn Jermstad, Knut Tomas Dalen, Ragnhild Eskeland, , Mads Lerdrup

The paper maps H2A.Z incorporation during mouse oogenesis, identifying distinct patterns and their relationship with epigenetic marks and transposable elements.

Nature Communications · Dec 2, 2025Peer-reviewedDevelopmental Biology

LSD1 represses a neonatal/reparative gene program in adult intestinal epithelium

Rosalie T. Zwiggelaar, Håvard T. Lindholm, Madeleine Fosslie, Marianne Terndrup Pedersen, Yuki Ohta, Alberto Díez-Sánchez, Mara Martín-Alonso, Jenny Ostrop, Mami Matano, Naveen Parmar, Emilie Kvaløy, Roos R. Spanjers, Kamran Nazmi, Morten Beck Rye, Finn Drabløs, , Menno J. Oudhoff

This study identifies LSD1 as a repressor of a neonatal/reparative gene program in adult intestinal epithelium, showing its role in Paneth cell differentiation and regenerative capacity.

Science Advances · Sep 11, 2020Peer-reviewedDevelopmental Biology

Intestinal-epithelial LSD1 controls cytoskeletal-mediated cell identity including goblet cell effector responses required for gut inflammatory and infectious diseases

Naveen Parmar, Kyle Burrows, Håvard T. Lindholm, Rosalie T. Zwiggelaar, Mara Martín-Alonso, Madeleine Fosslie, Bruce Vallance, John Arne Dahl, Colby Zaph, Menno J. Oudhoff

This study shows that intestinal-epithelial LSD1 deletion leads to increased pathogenesis in inflammatory and infectious disease models by impairing cytoskeletal organization and goblet cell effector responses.

bioRxiv · Jul 9, 2020Immunology