Author

L. Avalle

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 3 Research Focus:differential expression· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

A. Savino· 2 shared E. Monteleone· 2 shared P. Provero· 2 shared V. Poli· 2 shared A. Byron· 1 shared A. E. P. Loftus· 1 shared A. Griffa· 1 shared A. Nguyen Phuong· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

A. Savino

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A novel ILK/STAT3 pathway controls plasticity in a neural stem cell model of glioblastoma.

A. E. P. Loftus, M. S. Romano, A. Nguyen Phuong, M. T. Muir, J. C. Dawson, L. Avalle, A. T. Douglas, R. L. Mort, A. Byron, N. O. Carragher, S. M. Pollard, V. G. Brunton, M. C. Frame

This study identifies a novel ILK-STAT3 pathway that regulates phenotypic plasticity and mesenchymal state in mouse glioblastoma stem cells.

bioRxiv · Jul 19, 2023 · PreprintCancer Biology

Network analysis allows to unravel breast cancer molecular features and to identify novel targets

A. Savino, L. Avalle, E. Monteleone, I. Miglio, A. Griffa, G. Accetta, P. Provero, V. Poli

This study uses network analysis on breast cancer gene expression data to identify molecular features and potential drug targets, particularly in basal-like breast cancer.

bioRxiv · Mar 6, 2019 · PreprintCancer Biology

SP1 and STAT3 functionally synergize to induce the RhoU small GTPase and a subclass of non-canonical WNT responsive genes correlating with poor prognosis in breast cancer.

E. Monteleone, V. Orecchia, P. Corrieri, D. Schiavone, L. Avalle, E. Moiso, A. Savino, I. Molineris, P. Provero, V. Poli

This study identifies SP1 and STAT3 cooperation in inducing RhoU and non-canonical WNT responsive genes linked to poor breast cancer prognosis.

bioRxiv · Aug 9, 2018Cancer Biology