Author

L. Ciallella

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Levi· 1 shared A. Rolandelli· 1 shared C. Verollet· 1 shared D. J. Palmero· 1 shared D. Palmero· 1 shared F. Fuentes· 1 shared G. Lugo Villarino· 1 shared J. Barros· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

A. Levi

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2 papers

Recent·Most cited

Elevated glycolytic metabolism of monocytes limits the generation of HIF-1α-driven migratory dendritic cells in tuberculosis

M. Maio, J. Barros, M. Joly, Z. Vahlas, J. L. Marin Franco, M. Genoula, S. Monard, M. B. Vecchione, F. Fuentes, V. Gonzalez Polo, M. F. Quiroga, M. Vermeulen, T. P. Vu Manh, R. J. Argüello, S. Inwentarz, , L. Balboa

This study shows that elevated glycolytic metabolism in monocytes during tuberculosis impairs their differentiation into migratory dendritic cells, limiting immune response.

bioRxiv · Apr 3, 2023Immunology

Immunosuppressive role of PGE2 during human tuberculosis

J. M. Pellegrini, N. L. Tateosian, M. P. Morelli, A. Rolandelli, N. O. Amiano, D. J. Palmero, A. Levi, L. Ciallella, M. I. Colombo, V. E. Garcia

This study shows PGE2 suppresses immune responses in human tuberculosis by inhibiting lymphoproliferation, cytokine production, and immunological receptor expression, while promoting autophagy in monocytes.

bioRxiv · Aug 4, 2020 · PreprintImmunology