Author

L. Scorrano

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. Georgakilas· 1 shared A. Kotsinas· 1 shared A. Nardin· 1 shared A. Polyzos· 1 shared A. Yeats· 1 shared C. Beninca· 1 shared C. Glytsou· 1 shared C. Lukas· 1 shared

Activity by Year

2017-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperCancer Biology

A. Georgakilas

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Activation of Ca2+ phosphatase Calcineurin regulates Parkin translocation to mitochondria and mitophagy

E. Marchesan, A. Nardin, S. Mauri, S. Di Paola, M. Chinellato, S. von Stockum, J. Chakraborty, S. Herkenne, V. Basso, E. Schrepfer, O. Marin, L. Cendron, D. L. Medina, L. Scorrano, E. Ziviani

This study identifies Calcineurin as a regulator of Parkin translocation to mitochondria and mitophagy, showing its role in both basal and stress-induced processes.

bioRxiv · Jan 31, 2023 · PreprintCell Biology

Opa1 overexpression protects from early onset Mpv17-/--related mouse kidney disease

M. Zeviani, M. Luna-Sanchez, C. Beninca, R. Cerutti, A. Yeats, L. Scorrano, C. Viscomi, G. Brea Calvo

This study shows that moderate Opa1 overexpression in Mpv17-/- mice delays kidney disease progression by preserving mitochondrial structure and function.

bioRxiv · Mar 18, 2020Pathology

Mutational signatures reveal the role of RAD52 in p53-independent p21 driven genomic instability

P. Galanos, G. Pappas, A. Polyzos, A. Kotsinas, I. Svolaki, N. Giakoumakis, C. Glytsou, I. Pateras, U. Swain, V. Souliotis, A. Georgakilas, N. Geacintov, L. Scorrano, C. Lukas, J. Lukas, , V. Gorgoulis

This study shows chronic p21WAF1/Cip1 expression causes genomic instability by suppressing DNA repair pathways, revealing Rad52 as a key player in this process.

bioRxiv · Sep 28, 2017Cancer Biology