Author

Levi W. Barse

Northwestern University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2

Frequent Co-Authors

Jennifer A. KearneyiD· 2 shared Nicole A. HawkinsiD· 2 shared Alfred L. GeorgeiD· 1 shared Andrew M. Chiu· 1 shared Anis ContractoriD· 1 shared Antonio Sanz-ClementeiD· 1 shared Christopher H. ThompsoniD· 1 shared Conor J. Dixon· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersNeuroscience

Jennifer A. KearneyORCID

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3 papers

Recent·Most cited

Altered neurological and neurobehavioral phenotypes in a mouse model of the recurrent KCNB1-p.R306C voltage-sensor variant

Seok Kyu Kang, Nicole A. Hawkins, Christopher H. Thompson, Erin M. Baker, Dennis M. Echevarria-Cooper, Levi W. Barse, Tyler Thenstedt, Conor J. Dixon, Nathan Speakes, Alfred L. George, Jennifer A. Kearney

This paper generates a Kcnb1R306C mouse model to study the effects of a voltage-sensing mutation in KCNB1, finding altered neurological and neurobehavioral phenotypes.

Neurobiology of Disease · Mar 13, 2024Peer-reviewedNeuroscience

Gabra2 is a genetic modifier of Dravet syndrome in mice

Nicole A. Hawkins, Toshihiro Nomura, Samantha Duarte, Levi W. Barse, Robert W. Williams, Gregg E. Homanics, Megan K. Mulligan, Anis Contractor, Jennifer A. Kearney

This study identifies Gabra2 as a genetic modifier of Dravet syndrome in mice by showing that a deletion in Gabra2 worsens seizure and survival phenotypes in Scn1a+/- mice, which is reversed by repairing the deletion.

Mammalian Genome · Jun 4, 2021Peer-reviewedGenetics

NMDAR-activated PP1 dephosphorylates GluN2B to modulate NMDAR synaptic content.

Andrew M. Chiu, Jiejie Wang, Michael Patrick Fiske, Pavla Hubálková, Levi W. Barse, John A. Gray, Antonio Sanz-Clemente

The paper identifies phosphorylation of GluN2B at S1480 as a mechanism controlling NMDAR synaptic content through PP1-mediated dephosphorylation.

Cell Reports · Jul 1, 2019Peer-reviewedNeuroscience