Author

Lihang Yao

Merck & Co., Inc., Rahway, NJ, USA (United States)

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Organism:human· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:database / resource· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:metabolomics· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

Nathan Galen HatcheriD· 3 shared Alice PrigentiD· 1 shared Bang-Lin WaniD· 1 shared Brenna BeezholdiD· 1 shared Caine Smith· 1 shared Casey L. Mahoney-CraneiD· 1 shared Cheryl E. G. LeynsiD· 1 shared Drèson L. RusselliD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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3 shared papersNeuroscience

Nathan Galen HatcherORCID

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Author profile →

3 papers

Recent·Most cited

Quantitative Imaging and Characterization of Ganglioside Diversity in Mouse and Human Brain Tissues

Shadrack Mulinge Mutuku, Nicolas R Tomasiello, Caine Smith, Greg Trevor Sutherland, Reza Ranjbar Choubeh, Maria José Q. Mantas, Lihang Yao, Nathan Galen Hatcher, Nico Verbeeck, Shane R. Ellis, Kim Ekroos

This study creates a spatial ganglioside atlas for mouse and human brains using mass spectrometry imaging, revealing species-specific distributions and differences between species and brain regions.

bioRxiv · May 24, 2026 · PreprintNeuroscience

Glucocerebrosidase activity and lipid levels are related to protein pathologies in Parkinson's disease

Cheryl E. G. Leyns, Alice Prigent, Brenna Beezhold, Lihang Yao, Nathan Galen Hatcher, Peining Tao, John J. Kang, EunRan R. Suh, Vivianna M. Van Deerlin, John Q. Trojanowski, Virginia M.‐Y. Lee, Matthew E Kennedy, Matthew J. Fell, Michael X. Henderson

The paper examines the relationship between glucocerebrosidase activity, lipid levels, and protein pathologies in Parkinson's disease and dementia with Lewy bodies.

npj Parkinson s Disease · May 11, 2023Peer-reviewedNeuroscience

Neuronopathic GBA1 L444P mutation accelerates glucosylsphingosine levels and formation of hippocampal alpha-synuclein inclusions

Casey L. Mahoney-Crane, Megha Viswanathan, Drèson L. Russell, Rachel A.C. Curtiss, Jennifer Freire, Sai Sumedha Bobba, Sean D. Coyle, Monika Kandebo, Lihang Yao, Bang-Lin Wan, Nathan Galen Hatcher, Sean M. Smith, Jacob Marcus, Laura A. Volpicelli‐Daley

This study shows that the GBA1 L444P mutation increases glucosylsphingosine levels and hippocampal alpha-synuclein inclusions in mice, contributing to synucleinopathy pathology.

Journal of Neuroscience · Dec 7, 2022Peer-reviewedNeuroscience