Author

Lya Parres

Centre international de recherche sur le cancer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Akram GhantousiD· 1 shared Aurèlie Sallé· 1 shared Caroline P. PoubeliD· 1 shared Christine CarreiraiD· 1 shared Claire RenardiD· 1 shared Cyrille Cuenin· 1 shared Ekaterina Bourova-FliniD· 1 shared Erika C. CossetiD· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Akram GhantousORCID

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1 paper

Recent·Most cited

Disruption of the epigenetic regulator BAP1 drives chromatin remodeling leading to the emergence of cells with breast cancer stem cell properties and aberrant glycosylation.

Mariana Gomes da Silva Araujo, Aurèlie Sallé, Vincent Cahais, Claire Renard, Cyrille Cuenin, Caroline P. Poubel, Stéphane Keïta, Thorsten Mosler, Christine Carreira, Gabrielle Goldman Levy, Lya Parres, Ekaterina Bourova-Flin, Sophie Rousseaux, Saadi Khochbin, Akram Ghantous, , Rita Khoueiry

This study shows that disrupting BAP1, an epigenetic regulator, causes chromatin remodeling and leads to breast cancer stem cell-like properties and altered glycosylation.

bioRxiv · Dec 12, 2024 · PreprintCancer Biology