Author

M. A. Vanoni

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:Drosophila· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Brossa· 1 shared A. Saadeldin· 1 shared A. Salvaggio· 1 shared B. Bussolati· 1 shared C. Gellera· 1 shared C. Paiardi· 1 shared C. Visentin· 1 shared D. Grifoni· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. Brossa

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Goat PRP14 (gPRP14) has xeno-antigenic properties and works as a vaccine in preclinical models of cancer.

S. Minucci, B. Bussolati, P. Massa, A. Brossa, R. Ravasio, M. Saadeldin, G. Degani, E. Papadimitriou, A. Saadeldin, A. Salvaggio, C. Visentin, G. Rizzi, S. Ricagno, L. Popolo, M. A. Vanoni, P. G. Pelicci

This study characterizes gPRP14 as a tumor-specific xeno-antigen that induces anti-tumor immunity in preclinical cancer models.

bioRxiv · Jul 4, 2022 · PreprintCancer Biology

Glutamine Synthetase-1 induces autophagy-lysosomal degradation of huntingtin aggregates and ameliorates animal motility in a Drosophila model for Huntington’s disease

L. Vernizzi, C. Paiardi, G. Licata, T. Vitali, S. Santarelli, M. Raneli, V. Manelli, M. Rizzetto, M. Gioria, M. E. Pasini, D. Grifoni, M. A. Vanoni, C. Gellera, F. Taroni, P. Bellosta

This study shows that GS1 expression in Drosophila neurons reduces huntingtin aggregates and improves motility in a Huntington’s disease model by enhancing autophagy and inhibiting TOR signaling.

bioRxiv · Apr 30, 2019Cell Biology