Author

M. C. Ramello

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:differential expression· 1 Assay:flow cytometry· 1

Frequent Co-Authors

A. Gruppi· 3 shared C. L. Montes· 3 shared J. Tosello Boari· 3 shared C. Rodriguez· 2 shared E. V. Acosta Rodriguez· 2 shared F. Fiocca Vernengo· 2 shared M. Gorosito Serran· 2 shared W. Richer· 2 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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3 shared papersImmunology

A. Gruppi

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

CD39+ conventional CD4+ T cells with exhaustion traits and cytotoxic potential infiltrate tumors and expand upon CTLA-4 blockade

S. N. Bossio, C. Abrate, J. Tosello Boari, C. Rodriguez, F. Canale, M. C. Ramello, W. Richer, D. Rocha, C. Sedlik, A. Salomon, E. Borcoman, A. del Castillo, A. Gruppi, E. Fernandez, E. V. Acosta Rodriguez, C. L. Montes

The paper identifies CD39+ CD4+ T cells in tumors with exhaustion and cytotoxic traits, showing their expansion with CTLA-4 blockade and association with better survival in breast cancer.

bioRxiv · Jan 30, 2023Immunology

PD-L1hi plasmablasts limit the T cell response during the acute phase of parasite infections

M. Gorosito Serran, F. Fiocca Vernengo, L. Almada, C. G. Beccaria, P. F. Canete, J. Rocco Alegre, J. Tosello Boari, M. C. Ramello, E. Wehrens, Y. Cai, E. I. Zuniga, C. L. Montes, E. V. Acosta Rodriguez, I. A. Cockburn, C. G. Vinuesa, A. Gruppi

The paper identifies PD-L1hi plasmablasts that suppress T cell responses during acute parasite infections.

bioRxiv · Jun 10, 2020 · PreprintImmunology

IL-17RA-signaling modulates CD8+ T cell survival and exhaustion during Trypanosoma cruzi infection

J. Tosello Boari, C. L. Araujo Furlan, F. Fiocca Vernengo, C. Rodriguez, M. C. Ramello, M. C. Amezcua Vesely, M. Gorosito Serran, N. G. Nuñez, W. Richer, E. Piaggio, C. L. Montes, A. Gruppi, E. Acosta Rodriguez

This study shows that IL-17RA-signaling supports CD8+ T cell survival and function during Trypanosoma cruzi infection, with its absence leading to increased parasite load and reduced T cell responses.

bioRxiv · May 11, 2018Immunology