Author

M. Gingrich

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:differential expression· 1 Assay:electronic health records· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

C. Cabello-Verrugio· 1 shared C. G. Perry· 1 shared C. Henriquez-Olguin· 1 shared C. M. Monaco· 1 shared D. Al-Sajee· 1 shared E. A. Richter· 1 shared G. Martin· 1 shared I. A. Rebalka· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperPhysiology

C. Cabello-Verrugio

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Author profile →

2 papers

Recent·Most cited

Skeletal Muscle Mitochondrial Morphology Negatively Affected by Loss of Xin

G. Martin, D. Al-Sajee, M. Gingrich, R. Chattha, M. Akcan, C. M. Monaco, M. C. Hughes, C. G. Perry, I. A. Rebalka, M. A. Tarnopolsky, T. J. Hawke

This study shows that loss of Xin in mice leads to altered mitochondrial morphology and function in skeletal muscle, without affecting mitochondrial dynamics.

bioRxiv · Sep 14, 2023 · PreprintCell Biology

Phosphoproteomic identification of Xin as a novel requirement for skeletal muscle disuse atrophy

Z. Li, P. Jensen, J. Abrigo, C. Henriquez-Olguin, M. Gingrich, N. Rytter, L. Gliemann, E. A. Richter, T. Hawke, C. Cabello-Verrugio, M. R. Larsen, T. E. Jensen

This study identifies Xin as a novel protein involved in skeletal muscle disuse atrophy using phosphoproteomic analysis and confirms its regulation in mice and humans.

bioRxiv · May 28, 2019 · PreprintPhysiology