Author

M. Itoh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cell Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:imaging· 1 Approach:microscopy & imaging analysis· 1

Frequent Co-Authors

E. Shagimardanova· 1 shared H. Nakata· 1 shared J. Oyama· 1 shared K. Nakayama· 1 shared K. Node· 1 shared K. Usui· 1 shared K.-i. Arai· 1 shared O. Gusev· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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1 shared paperBioengineering

E. Shagimardanova

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3 papers

Recent·Most cited

Three-dimensional analysis and in vivo imaging for sperm release and transport in the murine seminiferous tubule

Y. Kanazawa, T. Omotehara, H. Nakata, T. Hirashima, M. Itoh

This study uses 3D analysis and in vivo imaging to examine sperm movement and transport in murine seminiferous tubules, revealing reversed luminal flow patterns.

bioRxiv · Jan 31, 2022Cell Biology

Development of a Tet-On inducible expression system for the anhydrobiotic cell line, Pv11

S. Tokumoto, Y. Miyata, K. Usui, R. Deviatiiarov, T. Ohkawa, S. Kondratieva, E. Shagimardanova, O. Gusev, R. Cornette, M. Itoh, Y. Hayashizaki, T. Kikawada

The paper develops a Tet-On inducible expression system for the desiccation-tolerant Pv11 cell line, demonstrating successful expression of reporter genes and a complex enzyme.

bioRxiv · May 29, 2020Bioengineering

2-Cl-C.OXT-A Stimulates Contraction through the Suppression of Phosphodiesterase Activity in Human Induced Pluripotent Stem Cell-derived Cardiac Organoids

T. Kitsuka, M. Itoh, S. Amamoto, K.-i. Arai, J. Oyama, K. Node, S. Toda, S. Morita, T. Nishida, K. Nakayama

This study shows that COA-Cl increases cardiac organoid contraction by suppressing phosphodiesterase activity, independent of S1P1R and A1R.

bioRxiv · Feb 18, 2019Pharmacology & Toxicology