Author

M. J. Naylor

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Organism:human· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

A. J. Hoy· 1 shared A. McFarland· 1 shared A. P. T.· 1 shared A. S. Cazet· 1 shared A. S. Don· 1 shared A. S. Mellick· 1 shared A. Swarbrick· 1 shared B. A. Varghese· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCancer Biology

A. J. Hoy

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2 papers

Recent·Most cited

Acetyl-CoA carboxylase 1-dependent lipogenesis drives breast cancer progression

K. Tan, T. Owen, H. P. McEwen, P. Simpson, A. J. Hoy, D. E. James, A. S. Don, M. J. Naylor

This study shows ACC1 drives breast cancer progression through lipogenesis, with ACC1 knockout reducing tumor growth and altering lipid profiles in both mouse models and human cells.

bioRxiv · Jul 20, 2023 · PreprintCancer Biology

Id proteins promote a cancer stem cell phenotype in triple negative breast cancer via negative regulation of Robo1

W. S. Teo, H. Holliday, N. Karthikeyan, A. S. Cazet, D. L. Roden, K. Harvey, C. V. Konrad, R. Murali, B. A. Varghese, A. P. T., C.-L. Chan, A. McFarland, S. Junankar, S. Ye, J. Yang, , R. Nair

This study shows that Id proteins promote a cancer stem cell phenotype in triple negative breast cancer by negatively regulating Robo1, leading to Myc activation.

bioRxiv · Mar 11, 2020 · PreprintCancer Biology