Author

M. Lisjak

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Approach:experimental assay· 1 Assay:imaging· 1 Research Focus:metabolism & bioenergetics· 1 Approach:microscopy & imaging analysis· 1 Organism:mouse· 1

Frequent Co-Authors

A. Gucek· 1 shared A. Li· 1 shared A. Prasnicka· 1 shared A. Sakanovic· 1 shared A. Verkhratsky· 1 shared B. Bozic· 1 shared B. Rituper· 1 shared D. C. Hay· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

A. Gucek

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Protein tyrosine phosphatase receptor kappa regulates glycolysis and de novo lipogenesis to promote hepatocyte metabolic reprogramming in obesity

E. H. Gilglioni, A. Li, W. Saint-Pierre Wijckmans, T.-K. Shen, I. Perez-Chavez, G. Hovhannisyan, M. Lisjak, J. Negueruela, V. Vandenbempt, J. Bauza-Martinez, J. M. Herranz, D. Ezerina, S. Demine, Z. Feng, T. Vignane, , E. N. Gurzov

This study identifies PTPRK's role in promoting hepatocyte metabolic reprogramming in obesity by regulating glycolysis and lipogenesis, with potential therapeutic implications for obesity-related liver diseases.

bioRxiv · Dec 1, 2023 · PreprintCancer Biology

Redistribution of cholesterol from vesicle to plasmalemma controls fusion pore geometry

B. Rituper, A. Gucek, M. Lisjak, U. Gorska, A. Sakanovic, S. Trkov Bobnar, E. Lasic, M. Bozic, P. S. Abbineni, J. Jorgacevski, M. Kreft, A. Verkhratsky, F. M. Platt, G. Anderluh, M. Stenovec, , R. Zorec

The paper shows that cholesterol redistribution from vesicles to the plasmalemma constricts the fusion pore, while depletion widens it, proposing a cholesterol-dependent mechanism for pore geometry regulation.

bioRxiv · Apr 6, 2020 · PreprintCell Biology