Author

M. N. Shokhirev

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. F. M. Pinto· 1 shared A. Williams· 1 shared B. Emu· 1 shared B. McDonald· 1 shared C. O'Connor· 1 shared C. Tsui· 1 shared C.-Y. Chung· 1 shared D. Chen· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

A. F. M. Pinto

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cancer BiologybioRxivOct 29, 2021

RAF conformational autoinhibition and 14-3-3 proteins promote paradoxical activation

G. Mendiratta, K. Abbott, Y.-C. Li…·E. C. Stites

The paper investigates paradoxical activation of RAF kinases, showing that 14-3-3 proteins enhance this effect by stabilizing RAF conformations and dimerization.

DOI 10.7554/elife.82739

statistical model

𝕏 PostBluesky

ImmunologybioRxivSep 3, 2020

Oxidized Lipids and CD36-Mediated Lipid Peroxidation in CD8 T Cells Suppress Anti-Tumor Immune Responses

S. Xu, O. Chaudhary, P. Rodriguez-Morales…·S. M. Kaech

This study identifies CD36-mediated lipid peroxidation driven by oxidized lipids as a mechanism suppressing anti-tumor CD8 T cell functions in murine tumors.

DOI 10.1016/j.immuni.2021.05.003

mousebulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivPreprintDec 19, 2019

Tuft cells restrain pancreatic tumorigenesis through paracrine eicosanoid signaling

K. E. DelGiorno, C.-Y. Chung, H. C. Maurer…·G. Wahl

This study shows that tuft cells suppress pancreatic tumorigenesis by producing anti-inflammatory prostaglandin D2, which inhibits stromal activation, and identifies their protective role in early disease progression.

DOI 10.1101/2019.12.19.882985

mousehuman

𝕏 PostBluesky