Author

M. Ruiz-Torres

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. C. Gladstein· 1 shared A. El-Bakkali· 1 shared A. Guajardo· 1 shared A. Losada· 1 shared A. Viera· 1 shared B. Salvador-Barbero· 1 shared B. Stanger· 1 shared C. Aguirre-Portoles· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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1 shared paperCancer Biology

A. C. Gladstein

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Author profile →

3 papers

Recent·Most cited

PP2A-B55alpha,delta phosphatase counteracts Ki67-dependent chromosome individualization during mitosis

M. Sanz-Flores, M. Ruiz-Torres, C. Aguirre-Portoles, A. El-Bakkali, B. Salvador-Barbero, C. Villarroya-Beltri, S. Ortega, D. Megias, D. W. Gerlich, M. Alvarez-Fernandez, M. Malumbres

The paper identifies PP2A-B55alpha,delta phosphatase as counteracting Ki67-dependent chromosome individualization during mitosis in mice.

bioRxiv · Nov 30, 2023 · PreprintCell Biology

p53 restoration in small cell lung cancer identifies a latent Cyclophilin-dependent necrosis mechanism

J. Acosta, Q. Li, N. F. Freebrug, N. Murali, G. P. Grothusen, M. Cicchini, H. Mai, A. C. Gladstein, K. M. Adler, K. R. Doerig, J. Li, M. Ruiz-Torres, K. L. Manning, B. Stanger, L. Busino, , D. M. Feldser

This study shows that p53 restoration in SCLC can induce necrosis via cyclophilin-dependent mechanisms, revealing subtype-specific responses and potential therapeutic targets.

bioRxiv · Jul 22, 2022Cancer Biology

PDS5 proteins regulate the length of axial elements and telomere integrity during male mouse meiosis

A. Viera, I. Berenguer, M. Ruiz-Torres, R. Gomez Lencero, A. Guajardo, J. L. Barbero, A. Losada, J. A. Suja

This study shows that PDS5A and PDS5B are functionally redundant in mouse meiosis, as their simultaneous depletion causes defects in axial element length and telomere integrity during prophase I.

bioRxiv · Sep 10, 2019Cell Biology