Author

M. Sathe

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:fluorescence / live-cell microscopy· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

J. A. Theriot· 2 shared A. Disanza· 1 shared A. S. Kennard· 1 shared C. K. Prinz· 1 shared E. C. Labuz· 1 shared G. Muthukrishnan· 1 shared G. Scita· 1 shared J. Rae· 1 shared

Activity by Year

2017-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

J. A. Theriot

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Post-injury hydraulic fracturing drives fissure formation in the zebrafish basal epidermal cell layer

A. S. Kennard, M. Sathe, E. C. Labuz, C. K. Prinz, J. A. Theriot

This study shows that zebrafish epidermis forms fissures post-injury due to hydraulic fracturing driven by osmotic pressure, with myosin II involvement and fluid clearance via macropinocytosis.

bioRxiv · May 21, 2022Cell Biology

Listeria monocytogenes Co-opts Caveolin-Mediated E-cadherin Trafficking and Macropinocytosis for Epithelial Cell-to-Cell Spread

P. Radhakrishnan, M. Sathe, J. A. Theriot

This study identifies E-cadherin ubiquitination and caveolin-mediated trafficking as mechanisms enabling Listeria monocytogenes to spread between epithelial cells.

bioRxiv · Apr 6, 2022 · PreprintMicrobiology

Small GTPases and BAR domain proteins regulate branched actin to make clathrin and dynamin independent endocytic vesicles

M. Sathe, G. Muthukrishnan, J. Rae, A. Disanza, M. Thattai, G. Scita, R. G. Parton, S. Mayor

This study identifies the molecular machinery regulating clathrin and dynamin-independent endocytic vesicles, showing the sequential recruitment of ARF1/GBF1 and CDC42, and the roles of BAR domain proteins and actin polymerization.

bioRxiv · Jul 31, 2017Cell Biology