Author

M. Tamura

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:yeast· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1

Frequent Co-Authors

A. H. Oda· 2 shared K. Ohta· 2 shared A. Ito· 1 shared A. Ozaki· 1 shared A. Yoneshige· 1 shared F. Mahayot· 1 shared F. Sofyantoro· 1 shared F. Takeuchi· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersCell Biology

A. H. Oda

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Granulomatous inflammatory responses are elicited in the liver of PD-1 knockout mice by de novo genome mutagenesis

I. Nagaretnam, A. Yoneshige, F. Takeuchi, A. Ozaki, M. Tamura, S. Suzuki, T. Shigeoka, A. Ito, Y. Ishida

This study shows that PD-1 knockout mice with genome mutations develop liver granulomatous inflammation, supporting PD-1's role in suppressing autoimmune responses to neoantigens.

bioRxiv · Aug 17, 2023 · PreprintImmunology

Rapamycin-sensitive mechanisms confine the growth of fission yeast below the temperatures detrimental to cell physiology

Y. Morozumi, F. Mahayot, Y. Nakase, J. X. Soong, S. Yamawaki, F. Sofyantoro, Y. Imabata, A. H. Oda, M. Tamura, S. Kofuji, Y. Akikusa, K. Ohta, K. Shiozaki

This study shows that rapamycin enables fission yeast to grow at high temperatures by inhibiting TORC1, suggesting that TORC1 normally restricts high-temperature growth rather than promoting it.

bioRxiv · May 4, 2023Cell Biology

Autotoxin-mediated voluntary triage in starved yeast community

A. H. Oda, M. Tamura, K. Kaneko, K. Ohta, T. S. Hatakeyama

The paper shows yeast communities use autotoxins to selectively eliminate non-tagged cells during starvation, enabling survival through voluntary triage.

bioRxiv · Oct 17, 2020 · PreprintMicrobiology