Author

Maria O. Levitin

Wellcome Sanger Institute

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetics· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1 Organism:non-human primate· 1

Frequent Co-Authors

Adam Hunter· 1 shared Alexandra S. NeaversoniD· 1 shared André GörgensiD· 1 shared Andrew Day· 1 shared Andrew H. CrosbyiD· 1 shared Andrew Y. ShueniD· 1 shared Angharad E. Evans· 1 shared Binnaz YalciniD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperGenetics

Adam Hunter

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2 papers

Recent·Most cited

Efficient genome editing in the non-human primate brain using programmable extracellular vesicles

Xiuming Liang, Houze Zhou, Zubair Ahmed Nizamudeen, Eleni Kyriakopoulou, Angharad E. Evans, Samantha Roudi, Hernando Yesid Estupiñan, Julia Rädler, Vincent Hou, Callum D.W. Daniel, Inés Hernández-Pérez, Malika Singh, Pardis Parsi, Daniel A. Lonergan, Lidia Menéndez Berlana, , Samir EL Andaloussi

This paper demonstrates efficient genome editing in non-human primate brains using engineered extracellular vesicles, achieving therapeutic potential for neurological disorders.

bioRxiv · Aug 6, 2026 · PreprintMolecular Biology

Mouse and cellular models of KPTN-related disorder implicate mTOR signalling in cognitive and progressive overgrowth phenotypes

Maria O. Levitin, Lettie E. Rawlins, Gabriela Sánchez-Andrade, Osama A. Arshad, Stephan C. Collins, Stephen J. Sawiak, Philip H. Iffland, Malin H. L. Andersson, Caleb P. Bupp, Emma L. Cambridge, Eve L. Coomber, Ian Ellis, Johanna C. Herkert, Holly Ironfield, Logan Jory, , Sebastian S. Gerety

The paper investigates KPTN-related disorder using mouse and human stem cell models, identifying mTOR pathway dysregulation as a key mechanism underlying cognitive and overgrowth phenotypes.

bioRxiv · Jul 15, 2022 · PreprintGenetics