Author

Max C. Overboom

Amsterdam University Medical Centers

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Assay:bulk RNA-seq· 1 Assay:organoid / primary culture· 1

Frequent Co-Authors

Peter Lodewijk HordijkiD· 3 shared Jan Willem BuikemaiD· 2 shared Philipp Christoph HaugeriD· 2 shared Albert C.W.A. van Wijk· 1 shared Carsten Tobias KuenneiD· 1 shared Fabienne PodiehiD· 1 shared Gabija Danilinaite· 1 shared Jisca MajoléeiD· 1 shared

Activity by Year

2022-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersCell Biology

Peter Lodewijk HordijkORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Marfan Patient iPSC-Derived Endothelial Cells Carrying FBN1 Variants Reveal Endothelial Dysfunction

Philipp Christoph Hauger, Gabija Danilinaite, Leonardo Spagnolello, Carsten Tobias Kuenne, Max C. Overboom, Jan Willem Buikema, Vivian de Waard, Peter Lodewijk Hordijk

This study uses iPSC-derived endothelial cells from Marfan syndrome patients to show impaired inflammatory resolution and endothelial dysfunction linked to FBN1 variants.

bioRxiv · Aug 21, 2026 · PreprintCell Biology

Vessel-on-Chip Model Of The Microcirculation In Abdominal Aortic Aneurysms

Philipp Christoph Hauger, Karlijn B. Rombouts, Marc Vila Cuenca, Albert C.W.A. van Wijk, Max C. Overboom, Jan Willem Buikema, Kak Khee Yeung, Valeria V. Orlova, Peter Lodewijk Hordijk

This study develops a patient-specific vessel-on-chip model of abdominal aortic aneurysms, showing preserved pathological features and pro-inflammatory responses in vitro.

Vascular Biology · Jan 1, 2026Peer-reviewedCell Biology

Rapid proteostasis controls monolayer integrity of quiescent endothelium

Fabienne Podieh, Roos Wensveen, Max C. Overboom, Lotte Abbas, Jisca Majolée, Peter Lodewijk Hordijk

The paper shows that rapid proteostasis is essential for maintaining monolayer integrity in quiescent endothelial cells by regulating protein turnover that affects cell-cell contact.

bioRxiv · Aug 9, 2022 · PreprintMolecular Biology