Philipp Christoph Hauger

This study uses iPSC-derived endothelial cells from Marfan syndrome patients to show impaired inflammatory resolution and endothelial dysfunction linked to FBN1 variants.

This paper develops a human iPSC-based model of the blood-brain barrier by co-culturing endothelial cells with pericytes and activating Wnt/β-catenin signaling, resulting in cells with BBB-like properties and gene expression patterns.

This study develops a patient-specific vessel-on-chip model of abdominal aortic aneurysms, showing preserved pathological features and pro-inflammatory responses in vitro.