Author

Monika Wierdl

St. Jude Children's Research Hospital

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

Lillian M. GuentheriD· 2 shared A Herman· 1 shared A Robichaud· 1 shared Adam David DurbiniD· 1 shared Alejandro Sweet-CorderoiD· 1 shared Anang A. ShelatiD· 1 shared Balakrishna KoneruiD· 1 shared Beisi XuiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersCancer Biology

Lillian M. GuentherORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Chemokine-Dependent Natural Killer Cells Prevent Pulmonary Metastasis in a Newly Established Mouse Osteosarcoma Model

Jifeng Yang, Frédérique Zindy, Shaela Fields, Judith W. Hyle, Laura J. Janke, Qianqian Li, Beisi Xu, Qiong Zhang, Gang Wu, Ti‐Cheng Chang, Monika Wierdl, Lillian M. Guenther, Linda M. Hendershot, Martine F. Roussel, Charles J. Sherr, Chunliang Li

This study identifies CCL2 as a chemokine that limits osteosarcoma metastasis via NK cell signaling in a mouse model.

bioRxiv · Jul 20, 2026 · PreprintCancer Biology

Tabular foundation model predicts alternative lengthening of telomeres (ALT) and identifies SMARCAL1 as a target in ALT-driven cancers

Declan Bennett, Monika Wierdl, S Chakraborty, Jordan Johnson, Victoria Akingbehin, Daniel Estevez-Prado, Yuan Feng, Janeala J. Morsby, John Harper, Mohammad Ali Mohammad Nezhady, M Pan, Nicole Ocasio-Martinez, Jerrel Lewis Catlett, TB Nations, Neekesh V. Dharia, , Lillian M. Guenther

The paper develops ALTitude, a machine learning model to predict ALT status from whole genome sequencing data, identifies SMARCAL1 as a therapeutic target in ALT-driven cancers.

bioRxiv · May 28, 2026 · PreprintCancer Biology