Author

N. C. Reich

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

V. Kirillov· 2 shared A. Chikhalya· 1 shared B. Liu· 1 shared B. Sheridan· 1 shared C. H. Hogan· 1 shared C. Khairallah· 1 shared G. V. Reynoso· 1 shared H. D. Hickman· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

V. Kirillov

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2 papers

Recent·Most cited

STAT3 is a genetic modifier of TGF-beta induced EMT in KRAS mutant pancreatic cancer

S. D'Amico, V. Kirillov, O. Petrenko, N. C. Reich

The paper identifies STAT3 as a genetic modifier of TGF-beta-induced EMT in KRAS mutant pancreatic cancer, showing its role in tumor maintenance and epithelial homeostasis.

eLife · Apr 4, 2024Peer-reviewedCancer Biology

B cell-intrinsic STAT3-mediated support of latency and interferon suppression during murine gammaherpesvirus 68 infection revealed through an in vivo competition model

C. H. Hogan, S. M. Owens, G. V. Reynoso, V. Kirillov, T. J. Meyer, M. A. Zelazowska, B. Liu, X. Li, A. Chikhalya, Q. Dong, C. Khairallah, N. C. Reich, B. Sheridan, K. M. McBride, P. Hearing, , L. T. Krug

This study shows that B cell-intrinsic STAT3 supports gammaherpesvirus latency and suppresses interferon responses in mice, using a competitive infection model.

bioRxiv · Mar 22, 2023 · PreprintMicrobiology