Author

N. L. Rosin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

J. Biernaskie· 2 shared A. Dufour· 1 shared A. Jaffer· 1 shared B. Surewaard· 1 shared B. Yipp· 1 shared C. M. Wood· 1 shared C. Robinson· 1 shared D. J. Mahoney· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCell Biology

J. Biernaskie

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Author profile →

2 papers

Recent·Most cited

The pore-forming apolipoprotein APOL7C drives phagosomal rupture and antigen cross-presentation by dendritic cells

G. A. Gonzales, S. Huang, J. Rajwani, L. Wilkinson, J. A. Nguyen, C. M. Wood, I. Dinh, M. Moore, E. Cedeno, S. Sikdar, N. McKenna, V. Ebacher, N. L. Rosin, M. B. Carneiro, B. Surewaard, , J. Canton

This study identifies APOL7C as a pore-forming protein that facilitates phagosomal rupture and antigen cross-presentation by cDC1s.

bioRxiv · Aug 11, 2023Cell Biology

SARS-CoV-2 infection of circulating immune cells is not responsible for virus dissemination in severe COVID-19 patients

N. L. Rosin, A. Jaffer, S. Sinha, R. P. Mulloy, C. Robinson, E. Labit, L. G. Almeida, A. Dufour, J. A. Corcoran, B. Yipp, J. Biernaskie

The paper investigates whether SARS-CoV-2 uses circulating immune cells to spread in severe COVID-19 patients and finds that these cells are not actively infected and not a source of viral dissemination.

bioRxiv · Jan 19, 2021 · PreprintImmunology