Author

Nanako Nakanishi

Mie University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Molecular Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Kazuaki MaruyamaiD· 2 shared Keiki NagaharuiD· 2 shared Eiichi MoriiiD· 1 shared Hidehito UsuiiD· 1 shared Kaoru DohiiD· 1 shared Katsutoshi HiroseiD· 1 shared Kyoko Imanaka-Yoshida· 1 shared Kyoko Imanaka‐YoshidaiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersPathology

Kazuaki MaruyamaORCID

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2 papers

Recent·Most cited

PathologyCardiovascular ResearchPeer-reviewedMar 17, 2026

VEGF-C-mediated Cardiac Lymphangiogenesis Promotes Inflammation Resolution in Autoimmune Acute Myocarditis in Mice

Nanako Nakanishi, Shigeru Hara, Keiki Nagaharu…·Kazuaki Maruyama

This study shows that VEGF-C treatment promotes cardiac lymphangiogenesis, reduces inflammation, and preserves heart function in autoimmune myocarditis in mice.

3 citationstop 4%

mousebulk RNA-seq

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Molecular BiologybioRxivPreprintOct 16, 2024

Embryological cellular origins and hypoxia-mediated mechanisms in PIK3CA-Driven refractory vascular malformations

Sota Torii, Keiki Nagaharu, Nanako Nakanishi…·Kazuaki Maruyama

This study identifies embryonic origins and hypoxia-driven mechanisms in PIK3CA-driven vascular malformations, showing that HIF-1 and VEGF-A inhibition reduces abnormalities.

DOI 10.1101/2024.10.16.618777

mousehuman

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