Author

Natali Neuhauser

Eötvös Loránd University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Organism:Drosophila· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

András RubicsiD· 2 shared Balint Solyom Botor· 2 shared Dorottya KárolyiiD· 2 shared Fergal O’FarrelliD· 2 shared Szabolcs TakátsiD· 2 shared Gabor E. BaloghiD· 1 shared Janka SzinyákovicsiD· 1 shared Mária PéteriD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersCell Biology

András RubicsORCID

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2 papers

Recent·Most cited

Autophagy promotes tumor growth through facilitating JAK/STAT signaling in a lysosomal degradation independent manner

András Rubics, Natali Neuhauser, Dorottya Károlyi, Balint Solyom Botor, Zhanerke Kenzhebayeva, Fergal O’Farrell, Szabolcs Takáts

This study shows that autophagy promotes tumor growth by enhancing JAK/STAT signaling independently of lysosomal degradation in Drosophila models.

Scientific Reports · Aug 10, 2026Peer-reviewedCell Biology

Fatty acid synthesis supports tumor progression through keeping TORC1 receptive for Insulin/PI3K signaling

Dorottya Károlyi, Balint Solyom Botor, Natali Neuhauser, András Rubics, Janka Szinyákovics, Tibor Kovács, Mária Péter, Gabor E. Balogh, Fergal O’Farrell, Szabolcs Takáts

The paper shows that fatty acid synthesis supports tumor progression by maintaining TORC1 sensitivity to Insulin/PI3K signaling through ACC-dependent mechanisms.

Cell Death and Disease · Apr 10, 2026Peer-reviewedCell Biology