Dorottya Károlyi

This study shows that autophagy promotes tumor growth by enhancing JAK/STAT signaling independently of lysosomal degradation in Drosophila models.

The paper shows that fatty acid synthesis supports tumor progression by maintaining TORC1 sensitivity to Insulin/PI3K signaling through ACC-dependent mechanisms.

This study identifies Hip14 and Patsas as rate-limiting factors in lysosome formation and fusion, linking defective palmitoylation to Huntington's disease-like symptoms in Drosophila.