Author

Nina Weinzierl

Heidelberg University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Abel Martín-Garrido· 2 shared Felix A. TrogischiD· 2 shared Gergana D. DobrevaiD· 2 shared Joerg HeinekeiD· 2 shared Julio CorderoiD· 2 shared Merve KeleşiD· 2 shared Norbert FreyiD· 2 shared Shruthi HemannaiD· 2 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

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2 shared papersPhysiology

Abel Martín-Garrido

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2 papers

Recent·Most cited

PhysiologybioRxivPreprintFeb 18, 2026

Loss of cardiomyocyte eukaryotic elongation factor 1A2 in mice triggers cardiomyopathy due to defective proteostasis

Abel Martín-Garrido, Nadine Weyrauch, Jorge Ruiz‐Orera…·Joerg Heineke

This study shows that loss of eEF1A2 in cardiomyocytes causes cardiomyopathy through proteostasis defects, and mTORC1 inhibition can reverse the condition.

DOI 10.64898/2026.01.14.699518

mouseproteomics

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Systems BiologyMolecular Therapy — Nucleic AcidsPeer-reviewedAug 15, 2024

Endothelial cell-derived, secreted long non-coding RNAs Gadlor1 and Gadlor2 aggravate pathological cardiac remodeling via intercellular crosstalk

Merve Keleş, Steve Grein, Natali Froese…·Joerg Heineke

This study identifies Gadlor1 and Gadlor2 as endothelial cell-derived lncRNAs that worsen cardiac remodeling through EV-mediated crosstalk with cardiomyocytes.

3 citations

mousebulk RNA-seq

𝕏 PostBluesky