Author

P. G. Morgan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1

Frequent Co-Authors

M. M. Sedensky· 3 shared S. C. Johnson· 3 shared J. Snell· 2 shared J. Stokes· 2 shared K. James· 2 shared K. Y. Park· 2 shared R. Bornstein· 2 shared A. Freed· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

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3 shared papersPathology

M. M. Sedensky

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Author profile →

3 papers

Recent·Most cited

Peripheral macrophages causally contribute to disease onset and progression in the Ndufs4(KO) model of Leigh syndrome

A. Hanaford, A. Khanna, K. James, Y. Chen, M. Mulholland, B. Kayser, V. Truong, M. M. Sedensky, P. G. Morgan, V. Kalia, S. Sarkar, S. C. Johnson

This study shows that peripheral macrophages contribute to CNS lesions in the Ndufs4(-/-) Leigh syndrome model, despite microglia depletion having limited effect.

bioRxiv · Jan 27, 2023 · PreprintPathology

Leukocyte proliferation mediates disease pathogenesis in the Ndufs4(KO) mouse model of Leigh syndrome

J. Stokes, R. Bornstein, K. James, K. Y. Park, K. Spencer, K. Vo, J. Snell, B. M. Johnson, P. G. Morgan, M. M. Sedensky, P. G. Morgan, N. A. Baertsch, S. C. Johnson

The paper shows that leukocyte proliferation drives Leigh syndrome pathology in Ndufs4(KO) mice, and leukocyte depletion prevents CNS lesions and extends survival.

bioRxiv · Nov 11, 2021 · PreprintPathology

Mechanisms underlying neonate specific metabolic effects of volatile anesthetics

J. Stokes, A. Freed, A. Pan, G. X. Sun, R. Bornstein, J. Snell, K. Y. Park, P. G. Morgan, M. M. Sedensky, S. C. Johnson

This study identifies a neonate-specific metabolic effect of volatile anesthetics, showing they deplete beta-hydroxybutarate at sub-anesthetic concentrations, mediated by citrate and malonyl-CoA changes, which adults are less sensitive to.

bioRxiv · Dec 8, 2020Physiology