Author

P. Whitfield

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cell Biology· 1

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Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Kar· 1 shared A. R. Barr· 1 shared A. T. Saurin· 1 shared C. Regnault· 1 shared J. A. Holt· 1 shared J. A. Marsh· 1 shared L. Crozier· 1 shared M. Badonyi· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

A. Kar

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1 paper

Recent·Most cited

Cell overgrowth during G1 arrest triggers an osmotic stress response and chronic p38 activation to promote cell cycle exit

L. Crozier, R. Foy, R. Adib, M. Badonyi, A. Kar, J. A. Holt, R. Wilson, C. Regnault, P. Whitfield, J. A. Marsh, A. T. Saurin, A. R. Barr, T. Ly

The paper shows that CDK4/6 inhibitor-induced G1 arrest causes cell overgrowth, triggering osmotic stress and p38-p53-p21 signaling to promote cell cycle exit.

bioRxiv · Sep 8, 2022Cell Biology