Author

Q. Mei

Primary ORCID profile (0000-0003-4648-7232 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Q. MeiiD1 paper · Rice University

Biology Disciplines

Click to filter author papers

Cancer Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

C. Herman· 2 shared D. Bates· 2 shared D. M. Fitzgerald· 2 shared J. P. Pribis· 2 shared J. Xia· 2 shared P. J. Hastings· 2 shared S. M. Rosenberg· 2 shared Y. Zhai· 2 shared

Activity by Year

2018-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersMicrobiology

C. Herman

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

PFKP regulates AXL-MET oncogenic and metabolic pathways in lung cancer

Y. Sun, H. Zhao, H. Feng, Y. Liu, Y. Li, S. Chen, Z. Zhou, Y. Du, X. Zeng, H. Ren, W. Su, Q. Mei, G. Chen

This study shows PFKP promotes lung cancer progression by interacting with the AXL-MET pathway, beyond its metabolic role in glycolysis.

bioRxiv · Mar 3, 2024 · PreprintCancer Biology

Gamblers: an Antibiotic-induced Evolvable Cell Subpopulation Differentiated by Reactive-oxygen-induced General Stress Response

J. P. Pribis, L. Garcia-Villada, Y. Zhai, O. Lewin-Epstein, A. Wang, J. Liu, J. Xia, Q. Mei, D. M. Fitzgerald, J. Bos, R. Austin, C. Herman, D. Bates, L. Hadany, P. J. Hastings, S. M. Rosenberg

The paper shows ciprofloxacin induces antibiotic-resistant mutations by creating a subpopulation of cells with activated sigma-S response, which allows error-prone repair and generates cross-resistant mutants.

bioRxiv · Dec 11, 2018Microbiology

Bacteria-to-human protein networks reveal origins of endogenous DNA damage

J. Xia, L.-Y. Chiu, R. B. Nehring, M. A. Bravo Nunez, Q. Mei, M. Perez, Y. Zhai, D. M. Fitzgerald, J. P. Pribis, Y. Wang, C. W. Hu, R. T. Powell, S. A. LaBonte, A. Jalali, M. L. Matadamas Guzman, , S. M. Rosenberg

The paper identifies DNA-damaging proteins in E. coli and their human homologs, showing they promote endogenous DNA damage and are overrepresented in cancer drivers.

bioRxiv · Jun 26, 2018Cancer Biology