Author

R. A. Sontz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1 Assay:proteomics· 1

Frequent Co-Authors

J. L. Merchant· 2 shared A. F. Diaz· 1 shared A. Schreibeis· 1 shared B. A. Wieland· 1 shared H. N. Thi Hong· 1 shared J. L. Chakrabarti· 1 shared L. L. Ding· 1 shared M. A. Helmrath· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCancer Biology

J. L. Merchant

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2 papers

Recent·Most cited

GFAP-directed Inactivation of Men1 Exploits Glial Cell Plasticity in Favor of Neuroendocrine Reprogramming

S. Duan, T. W. Sawyer, R. A. Sontz, B. A. Wieland, A. F. Diaz, J. L. Merchant

This study shows that inactivating Men1 in glial cells leads to neuroendocrine differentiation and tumorigenesis, exploiting glial plasticity.

bioRxiv · Feb 10, 2022Cancer Biology

Toll Like Receptor 9 Pathway Mediates Schlafen+-MDSC Polarization During Helicobacter-Induced Gastric Metaplasias

L. L. Ding, J. L. Chakrabarti, S. Sheriff, Q. Li, H. N. Thi Hong, R. A. Sontz, Z. E. Mendoza, A. Schreibeis, M. A. Helmrath, Y. Zavros, J. L. Merchant

This study shows that TLR9 mediates SLFN+-MDSC polarization during Helicobacter-induced gastric metaplasia, with the TLR9 C allele linked to increased risk of neoplastic complications.

bioRxiv · Jan 25, 2022Cancer Biology