Author

R. Eil

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1

Frequent Co-Authors

A. Bartlett· 1 shared A. J. Grossberg· 1 shared A. Q. Bartlett· 1 shared B. L. Worley· 1 shared C. Collier· 1 shared C. Jenkins· 1 shared H. Mendez· 1 shared J. Dickie· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

A. Bartlett

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

IL-6/STAT3 signaling drives early-stage pancreatic cancer cachexia via suppressed ketogenesis

P. C. Arneson-Wissink, H. Mendez, K. Pelz, J. Dickie, A. Q. Bartlett, B. L. Worley, S. M. Krasnow, R. Eil, A. J. Grossberg

This study identifies IL-6/STAT3 signaling as a driver of early-stage pancreatic cancer cachexia by suppressing ketogenesis and lipid oxidation in the liver.

Journal of Cachexia, Sarcopenia and Muscle · Apr 17, 2024Peer-reviewedCancer Biology

Intracellular K+ limits T cell exhaustion and preserves antitumor function

C. Collier, K. Wucherer, M. Mcwhorter, C. Jenkins, A. Bartlett, R. Roychoudhuri, R. Eil

This study shows that lowering intracellular K+ in T cells by disrupting Na+/K+ ATPase reduces T cell exhaustion and improves antitumor function by mitigating ROS accumulation.

bioRxiv · Sep 13, 2023Immunology