Author

R. T. Dirksen

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 1 Assay:electrophysiology· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1

Frequent Co-Authors

T. N. O'Connor· 2 shared A. Brasile· 1 shared A. F. Hazel· 1 shared A. R. Huber· 1 shared A. Ruffini· 1 shared B. F. Cravatt· 1 shared C. L. Cole· 1 shared C. Liang· 1 shared

Activity by Year

2018-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersPhysiology

T. N. O'Connor

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Voluntary wheel running mitigates disease in an Orai1 gain-of-function mouse model of tubular aggregate myopathy

T. N. O'Connor, N. Zhao, H. M. Orciuoli, A. Brasile, L. Pietrangelo, M. He, L. Groom, J. Leigh, Z. Mahamed, C. Liang, S. Malik, F. Protasi, R. T. Dirksen

Voluntary wheel running improves muscle function and reduces tubular aggregates in a mouse model of tubular aggregate myopathy.

bioRxiv · Sep 29, 2023Physiology

Glutathione supports lipid abundance in vivo

G. Asantewaa, E. T. Tuttle, N. P. Ward, Y. P. Kang, Y. Kim, M. E. Kavanagh, N. Girnius, Y. Chen, R. Duncan, K. Rodriguez, F. Hecht, M. Zocchi, L. Smorodintsev-Schiller, T. Q. Scales, K. Taylor, , I. S. Harris

This study shows that glutathione (GSH) is essential for lipid abundance in adult tissues, particularly in the liver, by repressing NRF2.

bioRxiv · Feb 10, 2023 · PreprintBiochemistry

Cardiac metabolic effects of KNa1.2 channel deletion, and evidence for its mitochondrial localization.

C. O. Smith, Y. T. Wang, S. M. Nadtochiy, J. H. Miller, E. A. Jonas, R. T. Dirksen, K. Nehrke, P. S. Brookes

This study identifies a cardiac mitochondrial KNa1.2 channel and shows its role in regulating metabolism during high energetic demand.

bioRxiv · Jan 23, 2018Cell Biology