Author

S. Balaji

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 3 Bioengineering· 2 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 7 Organism:mouse· 4 Research Focus:cell signaling & trafficking· 3 Research Focus:cell type annotation· 3 Research Focus:development & morphogenesis· 3 Assay:proteomics· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2

Frequent Co-Authors

B. W. Padon· 2 shared H. Li· 2 shared J. P. Jung· 2 shared O. S. Jung· 2 shared P. L. Bollyky· 2 shared T. J. Prajapati· 2 shared W. D. Short· 2 shared A. A. Kaul· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersBioengineering

B. W. Padon

Mutual co-authors in cluster

Author profile →

7 papers

Recent·Most cited

Unveiling Vaginal Fibrosis: A Novel Murine Model Using Bleomycin and Epithelial Disruption

J. M. McCracken, G. A. Calderon, L. Kumar, S. Balaji, F. Rivas, D. Erxleben, A. Hall, J. Hakim

This study develops a murine vaginal fibrosis model using bleomycin and epithelial disruption, showing fibrosis at 3 weeks with resolution by 6 weeks.

bioRxiv · Dec 18, 2023Physiology

Sustained ROS Scavenging and Pericellular Oxygenation by Lignin Composites Rescue HIF-1α and VEGF Levels to Improve Diabetic Wound Neovascularization and Healing

J. P. Jung, O. O. Olutoye, T. J. Prajapati, O. S. Jung, L. D. Yutzy, K. L. Nguyen, S. W. Wheat, J. Huang, B. W. Padon, F. Faruk, S. S. Keswani, P. Kogan, A. A. Kaul, L. Yu, H. Li, , S. Balaji

This paper develops lignosulfonate composites that enhance diabetic wound healing by promoting neovascularization and reducing ROS through sustained oxygen release and antioxidant properties.

bioRxiv · Jun 18, 2022Bioengineering

Engineering Antioxidant and Oxygen-Releasing Lignin Composites to Promote Wound Healing

S. Balaji, W. D. Short, B. W. Padon, J. A. Belgodere, S. E. Jimenez, N. T. Deoli, A. C. Guidry, J. C. Green, T. J. Prajapati, F. Farouk, A. Kaul, D. Son, O. S. Jung, C. E. Astete, M. Kim, J. P. Jung

This study develops lignin composites with oxygen-releasing nanoparticles to enhance wound healing through antioxidation and controlled oxygen delivery, showing improved tissue granulation and collagen remodeling.

bioRxiv · Mar 18, 2022Bioengineering

Caveolae Impacts Cellular RNA Levels through Transcription and Translational Processes

A. Qifti, S. Balaji, S. F. Scarlata

This study shows caveolae deformation affects protein levels and cytosolic RNA, impacting stress granule formation through cavin-1 relocalization.

bioRxiv · Apr 29, 2021 · PreprintPhysiology

The scaffold-dependent function of RIPK1 in dendritic cells promotes injury-induced colitis

K. Moriwaki, C. Park, K. Koyama, S. Balaji, K. Kita, R. Yagi, S. Komazawa-Sakon, T. Asuka, H. Nakano, Y. Kamada, E. Miyoshi, F. Chan

This study shows that RIPK1's scaffold function in dendritic cells prevents colonic inflammation and maintains immune homeostasis by inhibiting apoptosis and necroptosis.

bioRxiv · Feb 27, 2021Immunology

Filamentous Bacteriophage Delay Healing of Pseudomonas-Infected Wounds

M. S. Bach, C. R. de Vries, A. Khosravi, J. M. Sweere, M. Popescu, A. Hargil, J. D. Van Belleghem, G. Kaber, Q. Chen, E. B. Burgener, D. Liu, Q.-L. Tran, T. Dharmaraj, M. Birukova, V. Sunkari, , P. L. Bollyky

This study shows filamentous bacteriophage delays wound healing in Pseudomonas aeruginosa infections by inhibiting keratinocyte migration, supported by a patient cohort study.

bioRxiv · Mar 10, 2020Microbiology

IL-10 Promotes Endothelial Progenitor Cell Driven Wound Neovascularization and Enhances Healing via STAT3.

S. Balaji, e. steen, x. wang, H. V. Vangapandu, N. Templeman, A. J. Blum, C. M. Moles, H. Li, d. a. narmoneva, T. m. Crombleholme, m. j. butte, p. l. bollyky, s. g. keswani

This study shows IL-10 promotes wound healing by recruiting endothelial progenitor cells through STAT3-dependent regulation of VEGF and SDF-1 in both control and diabetic wounds.

bioRxiv · Sep 8, 2019 · PreprintPhysiology