Author

S. Winograd-Katz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:batch correction / integration· 1 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1 Assay:fluorescence / live-cell microscopy· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

B. Geiger· 3 shared A. Elson· 1 shared A. Kimchi· 1 shared A. M. Dowbaj· 1 shared A. Savidor· 1 shared C. Ampe· 1 shared E. Arman· 1 shared E. Sahai· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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3 shared papersCell Biology

B. Geiger

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Multi-site assessment of reproducibility in high-content live cell imaging data

J. Hu, X. Serra-Picamal, G.-J. Bakker, M. V. Troys, S. Winograd-katz, N. Ege, X. Gong, Y. Didan, I. Grosheva, O. Polansky, K. Bakkali, E. V. Hamme, M. v. Erp, M. Vullings, F. Weiss, , S. Stromblad

This study assesses reproducibility in high-content live cell imaging data across multiple sites and identifies sources of variability, showing that batch correction improves meta-analysis of perturbation experiments.

bioRxiv · Nov 18, 2022 · PreprintCell Biology

The Serine Protease HtrA2 mediates radiation-induced senescence in cancer cells

L. Hammer, V. Levin-Salomon, N. Yaeli-Slonim, M. Weiss, N. P. Dekel-Bird, T. Olender, Z. Porat, S. Winograd-Katz, A. Savidor, Y. Levin, S. Bialik, B. Geiger, A. Kimchi

This study identifies HtrA2 as a mediator of radiation-induced senescence in cancer cells, showing its role in proliferation arrest and vimentin filament reorganization.

bioRxiv · Feb 17, 2021Cancer Biology

R51Q SNX10 induces osteopetrosis by promoting uncontrolled fusion of monocytes to form giant, non-functional osteoclasts

M. Barnea, M. Stein, S. Winograd-Katz, M. Shalev, E. Arman, O. Brenner, F. Thalji, M. Kanaan, H. Elinav, P. Stepensky, B. Geiger, J. Tuckermann, A. Elson

This study identifies R51Q SNX10 mutation as a cause of osteopetrosis by promoting abnormal monocyte fusion into non-functional giant osteoclasts.

bioRxiv · Dec 24, 2018Cell Biology