Author

S. K. Barrick

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biophysics· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Research Focus:host-pathogen & immune response· 1 Research Focus:perturbation response prediction· 1 Approach:statistical model· 1

Frequent Co-Authors

M. J. Greenberg· 3 shared B. Scott· 1 shared J. A. Kirk· 1 shared L. Greenberg· 1 shared M. Papadaki· 1 shared S. A. Campbell· 1 shared S. P. Harris· 1 shared S. R. Clippinger· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersBiophysics

M. J. Greenberg

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Single Molecule Mechanics and Kinetics of Cardiac Myosin Interacting with Regulated Thin Filaments

S. R. Clippinger Schulte, B. Scott, S. K. Barrick, W. T. Stump, T. Blackwell, M. J. Greenberg

This study investigates how thin filament regulatory proteins affect the mechanics and kinetics of cardiac myosin using single-molecule techniques, finding no significant impact at physiological ATP concentrations but some effects at low ATP.

bioRxiv · Jan 9, 2023Biophysics

Myofilament Glycation in Diabetes Reduces Contractility by Inhibiting Tropomyosin Movement, is Rescued by cMyBPC Domains

M. Papadaki, T. Kampaengsri, S. K. Barrick, S. A. Campbell, S. P. Harris, M. J. Greenberg, J. A. Kirk

This study identifies myofilament glycation in diabetes as a cause of reduced cardiac contractility, showing that cMyBPC domains can rescue this dysfunction.

bioRxiv · Jun 9, 2021Biophysics

Computational tool to study perturbations in muscle regulation and its application to heart disease

S. K. Barrick, S. R. Clippinger, L. Greenberg, M. J. Greenberg

The paper introduces a computational tool to measure muscle activation equilibrium constants and analyze perturbations, applying it to study a mutation causing hypertrophic cardiomyopathy.

bioRxiv · Feb 19, 2019Biophysics