Author

S. R. Clippinger

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 4

Signature Assays, Organisms & Methods

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Approach:statistical model· 3 Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:benchmarking· 1 Approach:comparative / phylogenetic analysis· 1 Approach:deep learning· 1 Research Focus:perturbation response prediction· 1

Frequent Co-Authors

M. J. Greenberg· 4 shared L. Greenberg· 3 shared W. T. Stump· 3 shared P. E. Cloonan· 2 shared J. M. Nerbonne· 1 shared M. Ernst· 1 shared P. Angsutararux· 1 shared S. K. Barrick· 1 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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4 shared papersBiophysics

M. J. Greenberg

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4 papers

Recent·Most cited

Computational Tool for Ensemble Averaging of Single-Molecule Data

T. J. Blackwell, W. T. Stump, S. R. Clippinger, M. J. Greenberg

The paper introduces a computational tool, SPASM, for ensemble averaging of single-molecule data to improve the accuracy of measuring mechanochemical coupling in molecular motors.

bioRxiv · Aug 10, 2020Biophysics

Mechanical dysfunction induced by a hypertrophic cardiomyopathy mutation is the primary driver of cellular adaptation

S. R. Clippinger, P. E. Cloonan, W. Wang, L. Greenberg, W. T. Stump, P. Angsutararux, J. M. Nerbonne, M. J. Greenberg

This study shows that a HCM mutation causes mechanical dysfunction, which drives cellular adaptation through altered force generation and signaling pathways.

bioRxiv · May 4, 2020Biophysics

Disrupted mechanobiology links the molecular and cellular phenotypes in familial dilated cardiomyopathy

S. R. Clippinger, P. E. Cloonan, L. Greenberg, M. Ernst, W. T. Stump, M. J. Greenberg

This study investigates how a mutation in troponin-T affects cellular contractility and mechanosensing in familial dilated cardiomyopathy.

bioRxiv · Feb 21, 2019Biophysics

Computational tool to study perturbations in muscle regulation and its application to heart disease

S. K. Barrick, S. R. Clippinger, L. Greenberg, M. J. Greenberg

The paper introduces a computational tool to measure muscle activation equilibrium constants and analyze perturbations, applying it to study a mutation causing hypertrophic cardiomyopathy.

bioRxiv · Feb 19, 2019Biophysics