Author

S. Lantuejoul

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Assay:organoid / primary culture· 1

Frequent Co-Authors

A. Ballesta· 1 shared A. F. M. Dost· 1 shared A. Sexton-Oates· 1 shared A.-L. Huber· 1 shared A.-M. Dingemans· 1 shared B. Bartosch· 1 shared B. Guey· 1 shared B. Py· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. Ballesta

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2 papers

Recent·Most cited

Druggable Growth Dependencies and Tumor Evolution Analysis in Patient-Derived Organoids of Neuroendocrine Cancer

T. L. Dayton, N. Alcala, L. Moonen, L. den Hartigh, L. Mangiante, L. Lap, A. F. M. Dost, J. Beumer, S. Levy, R. S. van Leeuwaarde, W. M. Hackeng, K. Samsom, C. Voegele, A. Sexton-Oates, H. Begthel, , H. Clevers

This study establishes patient-derived tumor organoids (PDTOs) for neuroendocrine cancers, identifies therapeutic vulnerabilities, and highlights EGF dependency in pulmonary neuroendocrine tumors.

bioRxiv · Oct 31, 2022Cancer Biology

NLRP3 controls ATM activation in response to DNA damage.

V. PETRILLI, M. Bodnar-Wachtel, A.-L. Huber, J. Gorry, S. Hacot, L. Gerossier, B. Guey, N. Goutagny, B. Bartosch, E. Ballot, F. Ghiringhelli, B. Py, Y. Coute, A. Ballesta, S. Lantuejoul, J. Hall

This study shows NLRP3 interacts with ATM to regulate DNA damage response, and its loss reduces ATM activation and confers resistance to genomic stress in NSCLC.

bioRxiv · May 12, 2020Cancer Biology