Author

S. M. Ahmad

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Frustaci· 1 shared A. M. Shah· 1 shared A. Malik· 1 shared A. Muhee· 1 shared A. Protti· 1 shared B. Bhat· 1 shared C. Chimenti· 1 shared C. G. dos Remedios· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperPathology

A. Frustaci

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2 papers

Recent·Most cited

Scavenger receptor B1 facilitates the endocytosis of Escherichia coli via TLR4 signaling in mammary gland infection

Q. Taban, S. M. Ahmad, P. T. Mumtaz, B. Bhat, E. Haq, S. Magray, S. Saleeem, N. Shabir, A. Muhee, Z. A. Kashoo, M. Hameed, A. Malik, N. A. Ganai, R. A. Shah

This study shows SCARB1 facilitates E. coli endocytosis in goat mammary epithelial cells via TLR4 signaling during infection.

bioRxiv · Sep 5, 2022Immunology

Accumulation of prelamin A drives inflammation in the heart with implications for treatment of inherited and acquired cardiomyopathies

D. Brayson, A. Frustaci, R. Verardo, C. Chimenti, M. A. Russo, R. Hayward, S. M. Ahmad, G. Vizcay-Barrena, A. Protti, P. S. Zammit, C. G. dos Remedios, E. Ehler, A. M. Shah, C. M. Shanahan

This study shows prelamin A accumulation drives inflammation in both inherited and acquired cardiomyopathies, suggesting potential treatment strategies targeting inflammation.

bioRxiv · Nov 28, 2018Pathology