Author

E. Ehler

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Azad· 1 shared A. Frustaci· 1 shared A. M. Shah· 1 shared A. Protti· 1 shared C. Chimenti· 1 shared C. G. dos Remedios· 1 shared C. M. Shanahan· 1 shared D. Brayson· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

A. Azad

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2 papers

Recent·Most cited

Cardiomyocyte mechanical memory is regulated through the talin interactome and DLC1 dependent regulation of RhoA

E. Marhuenda, I. Xanthis, P. Pandey, A. Azad, M. Richter, D. Pavlovic, K. Gehmlich, G. Faggian, E. Ehler, J. Levitt, S. Ameer-Beg, T. Iskratsch

The paper identifies DLC1's role in regulating RhoA activity through the talin interactome, linking mechanical memory to cardiac phenotype changes.

bioRxiv · Jul 19, 2023 · PreprintCell Biology

Accumulation of prelamin A drives inflammation in the heart with implications for treatment of inherited and acquired cardiomyopathies

D. Brayson, A. Frustaci, R. Verardo, C. Chimenti, M. A. Russo, R. Hayward, S. M. Ahmad, G. Vizcay-Barrena, A. Protti, P. S. Zammit, C. G. dos Remedios, E. Ehler, A. M. Shah, C. M. Shanahan

This study shows prelamin A accumulation drives inflammation in both inherited and acquired cardiomyopathies, suggesting potential treatment strategies targeting inflammation.

bioRxiv · Nov 28, 2018Pathology