Author

G. Vizcay-Barrena

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Microbiology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:mouse· 1 Organism:plant (other)· 1

Frequent Co-Authors

A. Barnard· 1 shared A. Frustaci· 1 shared A. Hamacher-Brady· 1 shared A. M. Shah· 1 shared A. Protti· 1 shared A. Rueda-Zubiaurre· 1 shared A. Singh· 1 shared C. Chimenti· 1 shared

Activity by Year

2018-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperMicrobiology

A. Barnard

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Plasmodium falciparum protein Pfs16 is a target for transmission-blocking antimalarial drug development

S. Yahiya, C. N. Saunders, U. Straschil, O. J. Fischer, A. Rueda-Zubiaurre, S. Haase, G. Vizcay-Barrena, S. Jordan, S. Hassan, M. J. Delves, E. W. Tate, A. Barnard, M. J. Fuchter, J. Baum

This study identifies Pfs16 as the cellular target of the N-4HCS compound DDD01035881, which blocks Plasmodium male gamete formation.

bioRxiv · Jun 14, 2021Microbiology

Mitochondria form contact sites with the nucleus to couple pro-survival retrograde response

R. Desai, D. A. East, L. Hardy, M. Rigon, D. Faccenda, M. S. Alvarex, A. Singh, M. Mainenti, L. K. Hussey, R. Bentham, G. Szabadkai, V. Zappulli, G. Dhoot, L. E. Romano, X. Dong, , M. Campanella

The paper shows mitochondria form contact sites with the nucleus to facilitate a pro-survival retrograde response by stabilizing transcription factors like NF-kB.

bioRxiv · May 7, 2020 · PreprintCell Biology

Accumulation of prelamin A drives inflammation in the heart with implications for treatment of inherited and acquired cardiomyopathies

D. Brayson, A. Frustaci, R. Verardo, C. Chimenti, M. A. Russo, R. Hayward, S. M. Ahmad, G. Vizcay-Barrena, A. Protti, P. S. Zammit, C. G. dos Remedios, E. Ehler, A. M. Shah, C. M. Shanahan

This study shows prelamin A accumulation drives inflammation in both inherited and acquired cardiomyopathies, suggesting potential treatment strategies targeting inflammation.

bioRxiv · Nov 28, 2018Pathology