Author

S. M. Ali

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Physiology· 1 Plant Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:Arabidopsis· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. G. M. Ong· 1 shared A. Lipton· 1 shared A. M. Paul· 1 shared B. George· 1 shared D. P. F. Maffei· 1 shared E. Lopez-Juez· 1 shared G. N. Hortobagyi· 1 shared J. Bedard· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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1 shared paperPhysiology

A. G. M. Ong

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3 papers

Recent·Most cited

Mutations in TIC100 impair and repair chloroplast protein import and impact retrograde signalling

N. Loudya, D. P. F. Maffei, J. Bedard, S. M. Ali, P. F. Devlin, P. Jarvis, E. Lopez-Juez

Complementary mutations in TIC100 of the chloroplast inner envelope membrane cause reductions or corrective improvements in chloroplast protein import, and highlight a signalling role.

bioRxiv · Jan 18, 2022 · PreprintPlant Biology

Cellular Fitness Phenotypes of Cancer Target Genes from Oncobiology to Cancer Therapeutics

B. George, A. M. Paul, P. P. Mukundan, R. Amjesh, K. Leitzel, S. M. Ali, O. Sandiford, A. Lipton, P. Rameshwar, G. N. Hortobagyi, R. M. Pillai, R. Kumar

The paper examines fitness dependencies of cancer drug targets across 19 cancer types, finding that some targets are not fitness genes and suggesting drug repurposing opportunities.

bioRxiv · Jan 28, 2021 · PreprintCancer Biology

Protective functions of ZO-2/Tjp2 expressed in hepatocytes and cholangiocytes against liver injury and cholestasis

J. Xu, J. P. Kausalya, N. Van Hul, M. J. Caldez, S. Xu, A. G. M. Ong, W. L. Woo, S. M. Ali, P. Kaldis, W. Hunziker

This study shows that loss of Tjp2 in hepatocytes and cholangiocytes leads to cholestatic liver disease and increased susceptibility to liver injury, with protective roles in regulating bile acid transporters and detoxification enzymes.

bioRxiv · Nov 27, 2019Physiology