Author

S. M. Lewis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. Bera· 1 shared A. Cook· 1 shared A. E. Gallman· 1 shared A. Soldatenko· 1 shared C. J. Luckey· 1 shared C. Van Iderstine· 1 shared D. Liu· 1 shared E. Habib· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

A. Bera

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Innate and adaptive immunity to transfused allogenic red blood cells in mice requires MyD88

A. Soldatenko, L. R. Hoyt, L. Xu, S. Calabro, S. M. Lewis, A. E. Gallman, K. E. Hudson, S. R. Stowell, C. J. Luckey, J. C. Zimring, D. Liu, M. Santhanakrishnan, J. E. Hendrickson, S. C. Eisenbarth

This study shows that MyD88, not TRIF, is required for DC activation and alloantibody response to stored syngeneic RBCs in mice.

bioRxiv · Aug 24, 2021 · PreprintImmunology

Haploinsufficient tumour suppressor PRP4K is negatively regulated during epithelial-to-mesenchymal transition

L. E. Clarke, A. Cook, S. Mathavarajah, A. Bera, J. Salsman, E. Habib, C. Van Iderstine, M. Bydoun, S. M. Lewis, G. Dellaire

This study shows PRP4K is a haploinsufficient tumor suppressor whose levels are reduced during EMT, promoting invasion without full EMT in mammary cells.

bioRxiv · Apr 19, 2020Cancer Biology